Metabolic acidosis/Bibliography

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Books

• Gennari FJ, Adrogue HJ, Galla JH, Madias NE. (editors) (2005) Acid-Base Disorders and Their Treatment. Taylor & Francis Group: Boca Raton. ISBN 9780824759155. 856 pages.

• Rose BD, Post TW. (2001) Clinical Physiology of Acid-Base and Electrolyte Disorders. 5th ed. McGraw-Hill, Medical Pub. Division: New York. ISBN 0071346821.

Book chapters

• DuBose TD Jr. (2008) Disorders of Acid-Base Balance. Volume 1. Chapter 14. Page 505. In: Brenner BM (editor) (2008) Brenner & Rector's The Kidney. Eighth Edition. 2 volumes. Elsevier Inc., Saunders: Philadelphia. ISBN 978-1-4160-3105-5 (2-vol-set).

• Bevensee MO, Boron WF. (2008) Control of Intracellular pH. Volume 2. Chater 51. Page 1429. In: Alpern RJ, Hebert SC, Seldin DW, Giebisch GH. (editors) (2008) Seldin and Giebisch's The Kidney: Physiology & Pathophysiology. 2 volumes. Elsevier Inc., Academic Press: Amsterdam. ISBN 9780120884896. 2871 pages.

• Oh MS, Carroll HJ. (2008) External Balance of Electrolytes and Acids and Alkali. Volume 1. Chapter 10. Page 275. In: Alpern RJ, Hebert SC, Seldin DW, Giebisch GH. (editors) (2008) Seldin and Giebisch's The Kidney: Physiology & Pathophysiology. 2 volumes. Elsevier Inc., Academic Press: Amsterdam. ISBN 9780120884896. 2871 pages.

• Krapf R, Seldin DW, Alpern RJ. (2008) Clinical Syndromes of Metabolic Acidosis. Volume 2. Chapter 59. Page 1667. In: Alpern RJ, Hebert SC, Seldin DW, Giebisch GH. (editors) (2008) Seldin and Giebisch's The Kidney: Physiology & Pathophysiology. 2 volumes. Elsevier Inc., Academic Press: Amsterdam. ISBN 9780120884896. 2871 pages.

• Sebastian A, Frassetto LA, Morris RC Jr. (2008) The Acid-Base Effects of the Contemporary Western Diet: An Evolutionary Perspective. Volume 2. Page 1621. In: Alpern RJ, Hebert SC, Seldin DW, Giebisch GH. (editors) (2008) Seldin and Giebisch's The Kidney: Physiology & Pathophysiology. 2 volumes. Elsevier Inc., Academic Press: Amsterdam. ISBN 9780120884896. 2871 pages.

Journal articles

• Halperin ML, Kamel KS. (2010) Some observations on the clinical approach to metabolic acidosis. J Am.Soc.Nephrol. 21(6):894-897. PMID 20488948.
  • The traditional clinical analysis for the presence of metabolic acidosis suffers from a number of limitations that at times hinders one's ability to reach a proper diagnosis. Our aim here is to raise awareness of some of the nuanced difficulties and illustrate why other considerations add value.

• Adrogue NE, Madias NE. (2010) Secondary responses to altered acid-base status: the rules of engagement. J Am.Soc.Nephrol. 21(6):290-923. PMID 20431042.
  • Each of the four canonical acid-base disorders expresses as a primary change in carbon dioxide tension or plasma bicarbonate concentration followed by a secondary response in the countervailing variable. Quantified empirically, these secondary responses are directional and proportional to the primary changes, run a variable time course, and tend to minimize the impact on body acidity engendered by the primary changes. Absence of an appropriate secondary response denotes the coexistence of an additional acid-base disorder. Here we address the expected magnitude of the secondary response to each cardinal acid-base disorder in humans and offer caveats for judging the appropriateness of each secondary response.

• Gennari FJ, WeiseWJ. (2008) Acid-base disturbances in gastrointestinal disease (free full-text). Clin.J.Am.Soc.Nephrol. 3(6):1861-1868, PMID 18922984.
  • Disruption of normal gastrointestinal function as a result of infection, hereditary or acquired diseases, or complications of surgical procedures uncovers its important role in acid-base homeostasis. Metabolic acidosis or alkalosis may occur, depending on the nature and volume of the unregulated losses that occur. Investigation into the specific pathophysiology of gastrointestinal disorders has provided important new insights into the normal physiology of ion transport along the gut and has also provided new avenues for treatment. This review provides a brief overview of normal ion transport along the gut and then discusses the pathophysiology and treatment of the metabolic acid-base disorders that occur when normal gut function is disrupted.