Gut-brain signalling/Bibliography: Difference between revisions
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Näslund E. and Hellström P. M. (2007) Appetite signalling: From gut peptides and enteric nerves to brain. ''Physiology and behaviour'' 92:256-262. (''The signaling systems underlying eating behavior control are complex. The current review focuses on gastrointestinal (GI) signaling systems as physiological key functions for metabolic control. Many of the peptides that are involved in the regulation of food intake in the brain are also found in the enteric nervous system and enteroendocrine cells of the mucosa of the GI tract. Satiety signals from the GI tract act through the arcuate nucleus of the hypothalamus and the solitary tract nucleus of the brain stem, | Näslund E. and Hellström P. M. (2007) Appetite signalling: From gut peptides and enteric nerves to brain. ''Physiology and behaviour'' 92:256-262. (''The signaling systems underlying eating behavior control are complex. The current review focuses on gastrointestinal (GI) signaling systems as physiological key functions for metabolic control. Many of the peptides that are involved in the regulation of food intake in the brain are also found in the enteric nervous system and enteroendocrine cells of the mucosa of the GI tract. Satiety signals from the GI tract act through the arcuate nucleus of the hypothalamus and the solitary tract nucleus of the brain stem, | ||
where neuronal networks directly linked to hypothalamic centers for food intake and eating behavior are activated. We have primarily focused on GI effects of various gut peptides involved in the regulation of food intake, using motor activity as a biomarker for the understanding of gut peptide effects promoting satiety | where neuronal networks directly linked to hypothalamic centers for food intake and eating behavior are activated. We have primarily focused on GI effects of various gut peptides involved in the regulation of food intake, using motor activity as a biomarker for the understanding of gut peptide effects promoting satiety''.) | ||
==Primary Research Papers== | ==Primary Research Papers== | ||
#Dockray GJ. (2008) The versatility of the vagus. ''Physiology & Behaviour'' 97:531-536 (''Afferent neurons of the vagus nerve provide an important pathway for gut signals that act by triggering ascending pathways from the brain stem to hypothalamus...satiety hormone cholecystokinin (CCK) not only stimulates the discharge of these neurons but also controls their expression of both G-protein coupled receptors and peptide neurotransmitters known to influence food intake'') | #Dockray GJ. (2008) The versatility of the vagus. ''Physiology & Behaviour'' 97:531-536 (''Afferent neurons of the vagus nerve provide an important pathway for gut signals that act by triggering ascending pathways from the brain stem to hypothalamus...satiety hormone cholecystokinin (CCK) not only stimulates the discharge of these neurons but also controls their expression of both G-protein coupled receptors and peptide neurotransmitters known to influence food intake'') | ||
Revision as of 13:46, 12 October 2009
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Review Articles
- Magni P. et al. (2009) Feeding behavior in mammals including humans. Ann.N.Y.Acad.Sci. 1163:221-232. PMID 19456343
Bloom S. R. and Wren A. M. (2007) Gut hormomes and Appetite Control. Gastroenterology 132:2116-2130 (Many peptides are synthesized and released grom the gastrointestinal tract..they also physiologically influence eating behaviour...Ghrelin...peptide YY, pancreatic polypeptide, glucagon-like peptide 1, oxyntomodulin and cholecstokinin..)
Cowley M. A., Enriori P. J., Jobst E. E. (2004) The elctrophysiology of feeding circuits. Trends in Endocrinology and Metabolism 15,10:488-497 (The brain receives various sgnals that carry information about nutritional and metabolic status including neuropeptide PYY-3-36, ghrelin, cholecystokinin, leptin, glucose and insulin)
Schwartz M.W. et al. (2000) Central nervous system control of food intake. Nature 404:661-671. (New information regarding neuronal circuits that control food intake and their hormonal regulation has extended our understanding of energy homeostasis, the process whereby energy intake is matched to energy expenditure over time.)
Näslund E. and Hellström P. M. (2007) Appetite signalling: From gut peptides and enteric nerves to brain. Physiology and behaviour 92:256-262. (The signaling systems underlying eating behavior control are complex. The current review focuses on gastrointestinal (GI) signaling systems as physiological key functions for metabolic control. Many of the peptides that are involved in the regulation of food intake in the brain are also found in the enteric nervous system and enteroendocrine cells of the mucosa of the GI tract. Satiety signals from the GI tract act through the arcuate nucleus of the hypothalamus and the solitary tract nucleus of the brain stem, where neuronal networks directly linked to hypothalamic centers for food intake and eating behavior are activated. We have primarily focused on GI effects of various gut peptides involved in the regulation of food intake, using motor activity as a biomarker for the understanding of gut peptide effects promoting satiety.)
Primary Research Papers
- Dockray GJ. (2008) The versatility of the vagus. Physiology & Behaviour 97:531-536 (Afferent neurons of the vagus nerve provide an important pathway for gut signals that act by triggering ascending pathways from the brain stem to hypothalamus...satiety hormone cholecystokinin (CCK) not only stimulates the discharge of these neurons but also controls their expression of both G-protein coupled receptors and peptide neurotransmitters known to influence food intake)