Energostatic hypothesis/Bibliography
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Amanda Jayne Swan 16:40, 10 October 2011 (UTC) Amanda Jayne Swan 16:33, 10 October 2011 (UTC)
1. Andersson U, Filipsson K, Abbott CR, Woods A, Smith K, Bloom SR, et al. AMP-activated protein kinase plays a role in the control of food intake. Journal of Biological Chemistry. 2004;279(13):12005-8.
2. Booth DA. POSTABSORPTIVELY INDUCED SUPPRESSION OF APPETITE AND ENERGOSTATIC CONTROL OF FEEDING. Physiology & Behavior. 1972;9(2):199-202.
Proposed that a common metabolic measure of energy, rather than a particular nutrient, controls eating and appetite.
3. Booth DA, Lovett D, Simson PC. SUBCUTANEOUS DIALYSIS IN STUDY OF EFFECTS OF NUTRIENTS ON FEEDING. Physiology & Behavior. 1970;5(10):1201-&.
4. Friedman MI. CONTROL OF ENERGY-INTAKE BY ENERGY-METABOLISM. American Journal of Clinical Nutrition. 1995;62(5):S1096-S100.
How eating behaviour is linked with energy metabolism. Changes in liver metabolism provide signals for satiety and hunger.
5. Havel PJ. Peripheral signals conveying metabolic information to the brain: Short-term and long-term regulation of food intake and energy homeostasis. Experimental Biology and Medicine. 2001;226(11):963-77.
6. Langhans W. Fatty acid oxidation in the energostatic control of eating-A new idea. Appetite. 2008;51(3):446-51.
7. Lemagnen J, Devos M. METABOLIC CORRELATES OF MEAL ONSET IN FREE FOOD INTAKE OF RATS. Physiology & Behavior. 1970;5(7):805-&.
8. Nicolaidis S, Even PC. THE ISCHYMETRIC CONTROL OF FEEDING. International Journal of Obesity. 1990;14:35-52
Suggested that the metabolic rate is metered in the brain to affect eating. Whenever locomotion-free metabolism (MF) was low, hunger was promoted. When MF levels were replenished, hunger was prevented.