Talk:Mitochondrion
High priority
This is a high priority Google search item--don't ask me why, we don't have an article about it yet! --Larry Sanger 16:58, 11 October 2007 (CDT)
- This is the third-most popular page on CZ according to Special:PopularPages. I wonder why? John Stephenson 10:47, 22 September 2010 (UTC)
About "Neurotoxicity and neuroprotection"
I think that the processes of necrosis and apoptosis involve free radical generation catalyzed by Ca++ (calcification of the mitochondrion). If I understood well, Ca++ in mitochondria behaves like Fe++ and Cu++ out of mitochondria: it catalyzes Fenton reactions.
Perhaps we could determine whether all this is restricted to neuronal mitochondria, or if all mitochondria eventually follow the same pathological/senescent course.
Pierre-Alain Gouanvic 01:43, 2 February 2008 (CST)
Updated external links
As you know from time to time external links expire. I have just ascertained that all external links in this article are still functioning and there is no current dead link. Thomas Simmons 20:49, 14 January 2009 (UTC)